Science Based Nutrition

March 9, 2010

Obesity as protection against metabolic syndrome, not its cause

Filed under: Main Content — Tags: , , , , , , , , — ScienceDaily: Nutrition News @ 3:00 am
The collection of symptoms that is the metabolic syndrome -- insulin resistance, high cholesterol, fatty liver, and a greater risk for diabetes, heart disease, and stroke -- are all related to obesity, but, according to a new review not in the way you probably think they are.

March 8, 2010

Intestinal bacteria drive obesity and metabolic disease in immune-altered mice

Mice lacking a gene called TLR5 have an altered ability to recognize and control bacteria in their intestines, leading them to develop obesity and insulin resistance, which is often referred to as "pre-diabetes." The bacteria appear to influence appetite and metabolism rather than how well calories are absorbed. Obesity and insulin resistance can be transferred from TLR5-deficient mice via intestinal bacteria.

February 13, 2010

Role of protein pair in obesity regulation

Filed under: Main Content — Tags: , , , — ScienceDaily: Nutrition News @ 3:00 am
New research implicates a new protein in obesity development and highlights a protein pair's "team effort" in regulating obesity and insulin resistance.

February 5, 2010

Molecular ‘firing squad’ in mice triggered by overeating destroys metabolism

Filed under: Main Content — Tags: , , , , , , , — ScienceDaily: Nutrition News @ 9:00 am
Overeating in mice triggers a molecule once considered to be only involved in detecting and fighting viruses to also destroy normal metabolism, leading to insulin resistance and setting the stage for diabetes.

February 3, 2010

Making macrophages protect against effects of obesity

It is well known that diet-induced obesity increases dramatically a person's risk of developing type 2 diabetes. One reason underlying this susceptibility is that diet-induced obesity triggers the accumulation of inflammatory immune cells known as macrophages in fat tissue known as white adipose tissue (WAT). Researchers have now determined that engineering macrophages to store increased amounts of triacylglycerol (the main constituent of vegetable oil and animal fats) is sufficient to protect mice from diet-induced inflammatory macrophage activation, macrophage accumulation in WAT, and insulin resistance, a condition that preempts the onset of type 2 diabetes.
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